Rho/Rok-dependent regulation of actomyosin contractility at tricellular junctions controls epithelial permeability in Drosophila DOI Open Access
Thea Jacobs, Jone Isasti Sanchez,

Steven Reger

et al.

bioRxiv (Cold Spring Harbor Laboratory), Journal Year: 2024, Volume and Issue: unknown

Published: Oct. 5, 2024

Summary Cell contacts in epithelia are remodeled to regulate paracellular permeability and control passage of migrating cells, but how barrier function is modulated while preserving epithelial integrity not clear. In the follicular epithelium Drosophila ovaries, tricellular junctions (TCJs) open transiently a process termed patency allow externally produced yolk proteins for uptake by oocyte. Here we show that modulation actomyosin contractility at cell vertices controls TCJ permeability. Before patency, circumferential bundles anchored apical follicle vertices, where tension-sensing junctional proteins, Rho-associated kinase (Rok), active Myosin II accumulate maintain closed. opening initiated redistribution from medial pool, accompanied decreasing tension on vertices. This transition requires activation Cofilin-dependent F-actin disassembly phosphatase Slingshot inactivation light chain phosphatase, counteracted Rok. Accordingly, constitutive or Rho signaling prevent vertex opening, whereas reduced Rok activity cause excessive premature opening. Thus, intercellular gaps does require actomyosin-based forces, relies reduction contractility. Conversely, assembly required closing after patency. Our findings consistent with force transduction model which maintained vertex-anchored contractile actomyosin. We propose cell-type-specific organization determines mode contractility-dependent regulation

Language: Английский

Rho/Rok-dependent regulation of actomyosin contractility at tricellular junctions restricts epithelial permeability in Drosophila DOI Creative Commons
Thea Jacobs, Jone Isasti Sanchez,

Steven Reger

et al.

Current Biology, Journal Year: 2025, Volume and Issue: unknown

Published: Feb. 1, 2025

Language: Английский

Citations

1

The dynamics of tubulogenesis in development and disease DOI
Adrián Romero, Brandy L. Walker, Vanja Krneta‐Stankic

et al.

Development, Journal Year: 2025, Volume and Issue: 152(3)

Published: Feb. 1, 2025

ABSTRACT Tubes are crucial for the function of many organs in animals given their fundamental roles transporting and exchanging substances to maintain homeostasis within an organism. Therefore, development maintenance these tube-like structures is a vital process. can form diverse ways, advances our understanding molecular cellular mechanisms underpinning different modes tubulogenesis have significant impacts biological contexts, including disease. This Review discusses recent progress developmental underlying tube formation.

Language: Английский

Citations

0

Neighbor cells restrain furrowing during Xenopus epithelial cytokinesis DOI
Jennifer Landino, Eileen Misterovich, Lotte van den Goor

et al.

Developmental Cell, Journal Year: 2025, Volume and Issue: unknown

Published: April 1, 2025

Language: Английский

Citations

0

Vertex remodeling during epithelial morphogenesis DOI Creative Commons
Kaoru Sugimura, Tetsuhisa Otani

Current Opinion in Cell Biology, Journal Year: 2024, Volume and Issue: 91, P. 102427 - 102427

Published: Sept. 26, 2024

Epithelial cells adhere to each other via intercellular junctions that can be classified into bicellular and tricellular contacts (vertices). morphogenesis involves cell rearrangement requires remodeling of vertices. Although our understanding how junction mechanics drive epithelial has advanced, the mechanisms underlying vertex during this process have only received attention recently. In review, we outline recent progress in reorganize adhesion cytoskeleton trigger displacement resolution We will also discuss achieve optimal balance between structural flexibility stability their Finally, introduce new modeling frameworks designed analyze at Integration live imaging techniques is providing insights active roles vertices morphogenesis.

Language: Английский

Citations

2

Rho/Rok-dependent regulation of actomyosin contractility at tricellular junctions controls epithelial permeability in Drosophila DOI Open Access
Thea Jacobs, Jone Isasti Sanchez,

Steven Reger

et al.

bioRxiv (Cold Spring Harbor Laboratory), Journal Year: 2024, Volume and Issue: unknown

Published: Oct. 5, 2024

Summary Cell contacts in epithelia are remodeled to regulate paracellular permeability and control passage of migrating cells, but how barrier function is modulated while preserving epithelial integrity not clear. In the follicular epithelium Drosophila ovaries, tricellular junctions (TCJs) open transiently a process termed patency allow externally produced yolk proteins for uptake by oocyte. Here we show that modulation actomyosin contractility at cell vertices controls TCJ permeability. Before patency, circumferential bundles anchored apical follicle vertices, where tension-sensing junctional proteins, Rho-associated kinase (Rok), active Myosin II accumulate maintain closed. opening initiated redistribution from medial pool, accompanied decreasing tension on vertices. This transition requires activation Cofilin-dependent F-actin disassembly phosphatase Slingshot inactivation light chain phosphatase, counteracted Rok. Accordingly, constitutive or Rho signaling prevent vertex opening, whereas reduced Rok activity cause excessive premature opening. Thus, intercellular gaps does require actomyosin-based forces, relies reduction contractility. Conversely, assembly required closing after patency. Our findings consistent with force transduction model which maintained vertex-anchored contractile actomyosin. We propose cell-type-specific organization determines mode contractility-dependent regulation

Language: Английский

Citations

0