Direct modulation of G protein-gated inwardly rectifying potassium (GIRK) channels DOI Creative Commons

Ha Nguyen,

Ian W. Glaaser, Paul A. Slesinger

et al.

Frontiers in Physiology, Journal Year: 2024, Volume and Issue: 15

Published: June 6, 2024

Ion channels play a pivotal role in regulating cellular excitability and signal transduction processes. Among the various ion channels, G-protein-coupled inwardly rectifying potassium (GIRK) serve as key mediators of neurotransmission responses to extracellular signals. GIRK are members larger family inwardly-rectifying (Kir) channels. Typically, activated via direct binding G-protein βγ subunits upon activation receptors (GPCRs). channel requires presence lipid signaling molecule, phosphatidylinositol 4,5-bisphosphate (PIP

Language: Английский

A critical review of ethanol effects on neuronal firing: A metabolic perspective DOI Creative Commons
Dina Popova, Jacquelyne Ka‐Li Sun, Hei‐Man Chow

et al.

Alcohol Clinical and Experimental Research, Journal Year: 2024, Volume and Issue: 48(3), P. 450 - 458

Published: Jan. 12, 2024

Abstract Ethanol metabolism is relatively understudied in neurons, even though changes neuronal are known to affect their activity. Recent work demonstrates that ethanol preferentially metabolized over glucose as a source of carbon and energy, it reprograms neurons state reduced energy potential diminished capacity utilize once exhausted. intake has been associated with firing specific brain activity (EEG) patterns have linked risk for alcohol use disorder (AUD). Furthermore, haplotype the inwardly rectifying potassium channel subunit, GIRK2, which plays critical role regulating excitability AUD shown be directly regulated by ethanol. At same time, overexpression GIRK2 prevents ethanol‐induced metabolic changes. Based on available evidence, we conclude mechanisms underlying effects novel target developing therapies AUD.

Language: Английский

Citations

4

Epilepsy in a mouse model of GNB1 encephalopathy arises from altered potassium (GIRK) channel signaling and is alleviated by a GIRK inhibitor DOI Creative Commons
Sophie Colombo, Haritha P. Reddy,

Sabrina Petri

et al.

Frontiers in Cellular Neuroscience, Journal Year: 2023, Volume and Issue: 17

Published: May 18, 2023

mutations in

Language: Английский

Citations

10

Direct modulation of G protein-gated inwardly rectifying potassium (GIRK) channels DOI Creative Commons

Ha Nguyen,

Ian W. Glaaser, Paul A. Slesinger

et al.

Frontiers in Physiology, Journal Year: 2024, Volume and Issue: 15

Published: June 6, 2024

Ion channels play a pivotal role in regulating cellular excitability and signal transduction processes. Among the various ion channels, G-protein-coupled inwardly rectifying potassium (GIRK) serve as key mediators of neurotransmission responses to extracellular signals. GIRK are members larger family inwardly-rectifying (Kir) channels. Typically, activated via direct binding G-protein βγ subunits upon activation receptors (GPCRs). channel requires presence lipid signaling molecule, phosphatidylinositol 4,5-bisphosphate (PIP

Language: Английский

Citations

3