Reducing Brain Edema Using Berotralstat, an Inhibitor of Bradykinin, Repurposed as Treatment Adjunct in Glioblastoma DOI Creative Commons
Richard E. Kast

Neuroglia, Journal Year: 2024, Volume and Issue: 5(3), P. 223 - 233

Published: July 2, 2024

Glioblastomas synthesize, bear receptors for, and respond to bradykinin, triggering migration proliferation. Since centrifugal into uninvolved surrounding brain tissue occurs early in the course of glioblastoma, this attribute defeats local treatment attempts is primary reason current treatments almost always fail. Stopping bradykinin-triggered would be a step closer control disease. The recent approval marketing an oral plasma kallikrein inhibitor, berotralstat (Orladeyo™), pending FDA similar drug, sebetralstat, now offers potential method for reducing bradykinin production at sites bradykinin-mediated glioblastoma migration. Both drugs are approved treating hereditary angioedema. They ideal repurposing as adjunct glioblastoma. Furthermore, it has been established that peritumoral edema, common problem during clinical generated large part by locally produced via action. edema consequent use corticosteroids both shorten survival Therefore, (i) inhibition, (ii) growth (iii) reduction, (iv) less corticosteroids, may service slowing disease progression. This paper recounts details past research on rationale with berotralstat.

Language: Английский

Potential Benefits of Adding Alendronate, Celecoxib, Itraconazole, Ramelteon, and Simvastatin to Endometrial Cancer Treatment: The EC5 Regimen DOI Creative Commons
Richard E. Kast

Current Issues in Molecular Biology, Journal Year: 2025, Volume and Issue: 47(3), P. 153 - 153

Published: Feb. 26, 2025

Metastatic endometrial cancer continues to be a common cause of death as 2024, even after maximal use all currently available standard treatments. To address this problem metastatic generally in 2025, the drug repurposing movement within oncology identifies medicines general medical that have clinical or preclinical experimental data indicating they interfere with inhibit specific growth driving element identified given cancer. The also uses from large scale vitro screens thousands drugs, looking for simple empirical inhibition type. This paper outlines showing five drugs practice meet these evidence criteria growth, EC5 regimen. regimen osteoporosis treatment drug, alendronate; analgesic celecoxib; antifungal itraconazole; sleep aid, ramelteon; and cholesterol lowering simvastatin. Side effects seen are usually minimal easily tolerated by patients.

Language: Английский

Citations

0

UBC4: A Repurposed Drug Regimen for Adjunctive Use During Bladder Cancer Treatment DOI Creative Commons
Richard E. Kast

Biomedicines, Journal Year: 2025, Volume and Issue: 13(3), P. 706 - 706

Published: March 13, 2025

After it has metastasized, bladder cancer, the malignant transformation of urothelium, continues to be a common cause death after maximal use all currently available standard treatments. To address this problem in 2025, drug repurposing movement within oncology aims identify medicines general medical care that have data indicating they can interfere or inhibit growth driving element been identified cancer. This paper now outlines extensive preclinical showing four drugs from practice meet these criteria—the melatonergic ramelteon, antidepressant fluoxetine, antibiotic dapsone, and analgesic celecoxib. is UBC4 regimen, meant as possible adjunct added treatments metastatic Three factors justify clinical pilot trial UBC4: (1) are usually well tolerated carry low risk harm, (2) commonly fatal outcome cancer once widely plus (3) strong database UBC inhibition by each individual outlined paper.

Language: Английский

Citations

0

Reducing Brain Edema Using Berotralstat, an Inhibitor of Bradykinin, Repurposed as Treatment Adjunct in Glioblastoma DOI Creative Commons
Richard E. Kast

Neuroglia, Journal Year: 2024, Volume and Issue: 5(3), P. 223 - 233

Published: July 2, 2024

Glioblastomas synthesize, bear receptors for, and respond to bradykinin, triggering migration proliferation. Since centrifugal into uninvolved surrounding brain tissue occurs early in the course of glioblastoma, this attribute defeats local treatment attempts is primary reason current treatments almost always fail. Stopping bradykinin-triggered would be a step closer control disease. The recent approval marketing an oral plasma kallikrein inhibitor, berotralstat (Orladeyo™), pending FDA similar drug, sebetralstat, now offers potential method for reducing bradykinin production at sites bradykinin-mediated glioblastoma migration. Both drugs are approved treating hereditary angioedema. They ideal repurposing as adjunct glioblastoma. Furthermore, it has been established that peritumoral edema, common problem during clinical generated large part by locally produced via action. edema consequent use corticosteroids both shorten survival Therefore, (i) inhibition, (ii) growth (iii) reduction, (iv) less corticosteroids, may service slowing disease progression. This paper recounts details past research on rationale with berotralstat.

Language: Английский

Citations

0