Enhancement of SARS-CoV-2 Infection via Crosslinking of Adjacent Spike Proteins by N-Terminal Domain-Targeting Antibodies DOI Creative Commons
Tina Lusiany, Tohru Terada, Jun-ichi Kishikawa

et al.

Viruses, Journal Year: 2023, Volume and Issue: 15(12), P. 2421 - 2421

Published: Dec. 13, 2023

The entry of SARS-CoV-2 into host cells is mediated by the interaction between spike receptor-binding domain (RBD) and angiotensin-converting enzyme 2 (ACE2). Certain human antibodies, which target N-terminal (NTD) at a distant epitope from cell binding surface, have been found to augment ACE2 enhance infection. Notably, these antibodies exert their effect independently antibody fragment crystallizable (Fc) region, distinguishing mode action previously described antibody-dependent infection-enhancing (ADE) mechanisms. Building upon previous hypotheses experimental evidence, we propose that NTD-targeting (NIEAs) achieve through crosslinking neighboring proteins. In this study, present refined structural models NIEA antigen-binding region (Fab)–NTD complexes, supported molecular dynamics simulations hydrogen–deuterium exchange mass spectrometry (HDX-MS). Furthermore, provide direct evidence confirming NTDs NIEAs. Collectively, our findings advance understanding mechanisms underlying NIEAs impact on

Language: Английский

Pseudotyped Viruses: A Useful Platform for Pre-Clinical Studies Conducted in a BSL-2 Laboratory Setting DOI Creative Commons

Sofiia N. Rizatdinova,

Alina E. Ershova, Irina V. Astrakhantseva

et al.

Biomolecules, Journal Year: 2025, Volume and Issue: 15(1), P. 135 - 135

Published: Jan. 15, 2025

The study of pathogenic viruses has always posed significant biosafety challenges. In particular, the highly requires methods with low biological risk but relatively high sensitivity and convenience in detection. recent years, pseudoviruses, which consist a backbone one virus envelope proteins another virus, have become most widely used tools for exploring mechanisms binding to cells, membrane fusion viral entry, as well screening libraries antiviral substances, evaluating potential neutralizing monoclonal antibodies, developing neutralization tests, therapeutic platforms. During outbreak severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), pseudotyped virus-based assays played pivotal role advancing our understanding virus-cell interactions its disease pathogenesis. Such facilitated search agents accelerated epidemiological studies on post-infection post-vaccination humoral immunity. This review focuses use pseudoviruses model large-scale applications enveloped viruses.

Language: Английский

Citations

1

NIEAs elicited by wild-type SARS-CoV-2 primary infection fail to enhance the infectivity of Omicron variants DOI Creative Commons
Qi Gui, Haiyan Wang, Congcong Liu

et al.

Virology Journal, Journal Year: 2025, Volume and Issue: 22(1)

Published: Feb. 24, 2025

SARS-CoV-2 infection widely induces antibody response targeting diverse viral proteins, including typical representative N-terminal domain (NTD), receptor-binding (RBD), and S2 subunit of spike. A lot NTD-, RBD-, S2-specific monoclonal antibodies (mAbs) have been isolated from COVID-19 convalescents, some which displaying potent activities to inhibit infection. However, a small portion NTD-specific mAbs elicited by wild-type (WT) primary could facilitate the virus entry into target cells in vitro, so called NTD-targeting infection-enhancing (NIEAs). To date, has evolved massive variants carrying various NTD mutations, especially recent Omicron BA.2.86 JN.1. In this study, we investigated whether these WT-NIEAs still enhance infectivity emerging variants. Nine novel with germline gene usage were identified 3 individuals, effectively enlarging available panel NIEAs. Bivalent binding NIEAs inter-spike contributed their activities. emerged before Omicron, but ineffective JN.1, was because changed antigenicity NTDs. Overall, data clearly demonstrated cross-reactivity pre-existed series variants, helping evaluate risk enhanced future. Additional 9 individuals infected SARS-CoV-2. This Fc-independent enhancement mediated divalent F(ab')2 not Changed led ineffectiveness WT-induced

Language: Английский

Citations

0

Enhancement of SARS-CoV-2 Infection via Crosslinking of Adjacent Spike Proteins by N-Terminal Domain-Targeting Antibodies DOI Creative Commons
Tina Lusiany, Tohru Terada, Jun-ichi Kishikawa

et al.

Viruses, Journal Year: 2023, Volume and Issue: 15(12), P. 2421 - 2421

Published: Dec. 13, 2023

The entry of SARS-CoV-2 into host cells is mediated by the interaction between spike receptor-binding domain (RBD) and angiotensin-converting enzyme 2 (ACE2). Certain human antibodies, which target N-terminal (NTD) at a distant epitope from cell binding surface, have been found to augment ACE2 enhance infection. Notably, these antibodies exert their effect independently antibody fragment crystallizable (Fc) region, distinguishing mode action previously described antibody-dependent infection-enhancing (ADE) mechanisms. Building upon previous hypotheses experimental evidence, we propose that NTD-targeting (NIEAs) achieve through crosslinking neighboring proteins. In this study, present refined structural models NIEA antigen-binding region (Fab)–NTD complexes, supported molecular dynamics simulations hydrogen–deuterium exchange mass spectrometry (HDX-MS). Furthermore, provide direct evidence confirming NTDs NIEAs. Collectively, our findings advance understanding mechanisms underlying NIEAs impact on

Language: Английский

Citations

4