L-Arginine and Intermittent Hypoxia Are Stress-Limiting Factors in Male Wistar Rat Models DOI Open Access
Natalia Kurhaluk, Олександр Лукаш, Piotr Kamiński

et al.

International Journal of Molecular Sciences, Journal Year: 2024, Volume and Issue: 25(22), P. 12364 - 12364

Published: Nov. 18, 2024

The aim of this study was to evaluate the combined effects L-arginine, intermittent hypoxia training (IHT), and acute stress on oxygen-dependent processes in rats, including mitochondrial oxidative phosphorylation, microsomal oxidation, intensity lipoperoxidation processes. In addition, our investigated how modulatory effect NO synthase mechanism concentration catecholamines (CA), such as adrenaline noradrenaline, their biosynthetic precursors (DOPA, dopamine) varies depending cholinergic (acetylcholine, Ach-acetylcholinesterase, AChE) status rats. This protective stress-limiting L-arginine impact IHT blood liver results showed that promoted maintenance NAD-dependent oxidation mitochondria, which detrimental compared succinate accompanied by depletion respiratory activity reserves under induced high concentrations CA. interdependence SC-dependent functional role substrate chain conditions using inhibitors revealed importance system. Administration during course prior exposure increased compensatory capacity organism. sympathoadrenal system stress-exposed early stages IHT, modulation CA observed with a concomitant increase processes, final increased, but there also an inhibition lipoperoxidation, particularly enhanced administration L-arginine. ACh decrease AChE at different adaptation (days 5, 10, 14). Thus, method significantly mobilises reserve through CA, ACh-AChE mediated nitric oxide.

Language: Английский

L-Arginine and Nitric Oxide in Vascular Regulation—Experimental Findings in the Context of Blood Donation DOI Open Access
Natalia Kurhaluk, Halyna Tkachenko

Nutrients, Journal Year: 2025, Volume and Issue: 17(4), P. 665 - 665

Published: Feb. 13, 2025

This narrative review provides an analysis of the role nitric oxide (NO) and its precursors, particularly L-arginine, in vascular regulation health, with emphasis on findings from our experimental research animal models. NO serves as a critical mediator function, contributing to vasodilation, blood flow, prevention thrombosis. As primary precursor NO, L-arginine is essential for maintaining endothelial integrity, modulating mitochondrial reducing oxidative damage. synthesises data contextualises these within physiological challenges faced by donors, such repeated donation associated stress. It examines effects supplementation respiration, lipid peroxidation, microsomal oxidation different conditions, including differences age, gender, dietary interventions. The mechanisms which enhances production, improves elasticity, alleviates dysfunction caused reduced bioavailability are also investigated. By integrating insights existing literature, this perspective potential address specific needs donors. highlights importance personalised nutritional approaches enhancing donor recovery resilience. In addition, assesses wider implications mitigating stress preserving function. interplay between bioavailability, factors, adaptation donors highlighted, along identification current knowledge gaps recommendations future research. presenting both original evidence synthesis article therapeutic promoting health context donation.

Language: Английский

Citations

1

L-Arginine and Intermittent Hypoxia Are Stress-Limiting Factors in Male Wistar Rat Models DOI Open Access
Natalia Kurhaluk, Олександр Лукаш, Piotr Kamiński

et al.

International Journal of Molecular Sciences, Journal Year: 2024, Volume and Issue: 25(22), P. 12364 - 12364

Published: Nov. 18, 2024

The aim of this study was to evaluate the combined effects L-arginine, intermittent hypoxia training (IHT), and acute stress on oxygen-dependent processes in rats, including mitochondrial oxidative phosphorylation, microsomal oxidation, intensity lipoperoxidation processes. In addition, our investigated how modulatory effect NO synthase mechanism concentration catecholamines (CA), such as adrenaline noradrenaline, their biosynthetic precursors (DOPA, dopamine) varies depending cholinergic (acetylcholine, Ach-acetylcholinesterase, AChE) status rats. This protective stress-limiting L-arginine impact IHT blood liver results showed that promoted maintenance NAD-dependent oxidation mitochondria, which detrimental compared succinate accompanied by depletion respiratory activity reserves under induced high concentrations CA. interdependence SC-dependent functional role substrate chain conditions using inhibitors revealed importance system. Administration during course prior exposure increased compensatory capacity organism. sympathoadrenal system stress-exposed early stages IHT, modulation CA observed with a concomitant increase processes, final increased, but there also an inhibition lipoperoxidation, particularly enhanced administration L-arginine. ACh decrease AChE at different adaptation (days 5, 10, 14). Thus, method significantly mobilises reserve through CA, ACh-AChE mediated nitric oxide.

Language: Английский

Citations

2