CALR regulates the proliferation and drug sensitivity of multiple myeloma cells through the BCL2 signaling pathway DOI
Yin Wang,

Xiaoli Hu,

Yaqi Song

et al.

Research Square (Research Square), Journal Year: 2025, Volume and Issue: unknown

Published: April 29, 2025

Abstract Background: Calreticulin (CALR) has been implicated in the genesis and progression of numerous tumors. Nevertheless, its impact on multiple myeloma (MM) remains ambiguous. This study aimed to explore effect CALR proliferation drug sensitivity MM cells delve into underlying mechanism. Methods: First, expression was measured. Subsequently, knockdown validated both vitro vivo. Finally, RNA sequencing utilized molecular mechanisms associated with knockdown. Results: We determined that is upregulated cells. Both vivo functional assays demonstrated attenuated proliferative capacity cells, heightened their bortezomib (BTZ), facilitated cell apoptosis. RNA-sequencing results indicated activates apoptosis pathway, mechanism potentially involving regulation BCL2 signaling pathway. Conclusion: This indicates MM. Targeting might be a potential strategy further enhance treatment efficacy for

Language: Английский

CALR regulates the proliferation and drug sensitivity of multiple myeloma cells through the BCL2 signaling pathway DOI
Yin Wang,

Xiaoli Hu,

Yaqi Song

et al.

Research Square (Research Square), Journal Year: 2025, Volume and Issue: unknown

Published: April 29, 2025

Abstract Background: Calreticulin (CALR) has been implicated in the genesis and progression of numerous tumors. Nevertheless, its impact on multiple myeloma (MM) remains ambiguous. This study aimed to explore effect CALR proliferation drug sensitivity MM cells delve into underlying mechanism. Methods: First, expression was measured. Subsequently, knockdown validated both vitro vivo. Finally, RNA sequencing utilized molecular mechanisms associated with knockdown. Results: We determined that is upregulated cells. Both vivo functional assays demonstrated attenuated proliferative capacity cells, heightened their bortezomib (BTZ), facilitated cell apoptosis. RNA-sequencing results indicated activates apoptosis pathway, mechanism potentially involving regulation BCL2 signaling pathway. Conclusion: This indicates MM. Targeting might be a potential strategy further enhance treatment efficacy for

Language: Английский

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