The Microbiota–Gut–Brain Axis: Key Mechanisms Driving Glymphopathy and Cerebral Small Vessel Disease DOI Creative Commons
Che Mohd Nasril Che Mohd Nassir, Muhammad Danial Che Ramli, Mazira Mohamad Ghazali

et al.

Life, Journal Year: 2024, Volume and Issue: 15(1), P. 3 - 3

Published: Dec. 24, 2024

The human microbiota constitute a very complex ecosystem of microorganisms inhabiting both the inside and outside our bodies, in which health maintenance disease modification are main regulatory features. recent explosion microbiome research has begun to detail its important role neurological health, particularly concerning cerebral small vessel (CSVD), disorder associated with cognitive decline vascular dementia. This narrative review represents state-of-the-art knowledge intimate, interplay between brain through gut-brain axis (GBA) emerging glymphatic system dysfunction (glymphopathy) circulating cell-derived microparticles (MPs) as mediators these interactions. We discuss how microbial dysbiosis promotes neuroinflammation, dysfunction, impaired waste clearance brain, critical factors pathogenesis CSVD. Further, we lifestyle that shape composition functionality microbiota, focusing on sleep modifiable risk factor disorders. presents from neuroscientific perspective establish future therapeutic avenues targeting improve reduce burden

Language: Английский

Microbial Influences on Amyotrophic Lateral Sclerosis: The Gut–Brain Axis and Therapeutic Potential of Microbiota Modulation DOI Open Access
Victòria Ayala,

Laia Fontdevila,

Santiago Rico-Rios

et al.

Sclerosis, Journal Year: 2025, Volume and Issue: 3(1), P. 8 - 8

Published: March 5, 2025

Background/Objectives: Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disorder characterized by the progressive degeneration of motor neurons. The gut microbiota, community microorganisms in digestive tract, has recently been implicated ALS pathogenesis through its influence on neuroinflammation and metabolic pathways. This review explores potential role microbiota metabolites progression investigates therapeutic approaches targeting microbiota. Methods: A comprehensive current literature was conducted to assess relationship between composition, microbial metabolites, patients. We searched for published reports ALS, emphasizing complex interplay dysbiosis, neuroinflammation, systemic metabolism. Special emphasis placed studies exploring short-chain fatty acids (SCFAs), bacterial amyloids (curli-like factors), neurotoxins such as β-methylamino-L-alanine (BMAA). liver–gut axis evaluated well. changes would sustain rationale strategies probiotics, prebiotics, fecal transplantation (FMT), dietary interventions. Results: patients exhibit reduced SCFA-producing bacteria an increase potentially pathogenic genera. Of note, different do not agree common patterns being linked supporting need further, more extensive studies. Dysbiosis sometimes correlates with inflammation disrupted liver function, amplifying neuroinflammatory responses. Key including SCFAs, amyloids, BMAA, may exacerbate neuron promoting protein misfolding, oxidative stress, neuroinflammation. Emerging strategies, probiotics FMT, show restoring balance, although clinical data remain limited. Conclusions: could modulate metabolism ALS. Microbiota-targeted therapies, interventions, represent promising avenues mitigating disease progression. Further research required validate these interventions large-scale, longitudinal develop personalized microbiota-based treatments tailored individual phenotypes.

Language: Английский

Citations

1

The microbial metabolite trimethylamine N-oxide and the kidney diseases DOI Creative Commons
Jian Su,

Xiangqi Wu,

Qi Wang

et al.

Frontiers in Cellular and Infection Microbiology, Journal Year: 2025, Volume and Issue: 15

Published: March 11, 2025

Trimethylamine N-oxide (TMAO), a metabolite, is co-metabolite produced by both gut microbiota and livers, originating from foods rich in choline or carnitine. Emerging evidence suggests that TMAO may play role the pathogenesis of various kidney diseases, including acute injury chronic disease. Research has demonstrated heightened levels are correlated with likelihood disease advancement cardiovascular incidents among individuals Furthermore, been observed to stimulate inflammation, oxidative stress, fibrosis animal models Mechanistically, contribute inhibiting autophagy, activating NLRP3 inflammasome, inducing mitochondrial dysfunction. Therefore, targeting represent promising therapeutic strategy for treatment diseases. Future studies needed further investigate develop TMAO-targeted therapies prevention

Language: Английский

Citations

0

Association between serum alpha-1-acid glycoprotein (AGP) levels and depression: The mediation effect of triglyceride-glucose (TyG) index DOI
Kwan Hyung Yi, Huan Liu

Journal of Affective Disorders, Journal Year: 2025, Volume and Issue: unknown

Published: April 1, 2025

Language: Английский

Citations

0

Molecular Mechanisms and Emerging Precision Therapeutics in the Gut Microbiota-Cardiovascular Axis DOI Open Access

Jhon Alexander Ponce Alencastro,

Diego Lucero, Ricardo Pérez-Solís

et al.

Cureus, Journal Year: 2025, Volume and Issue: unknown

Published: April 26, 2025

Language: Английский

Citations

0

Association Between Lifestyle Patterns and Abdominal Obesity with Biochemical and Inflammatory Biomarkers in Adolescents with Down Syndrome: The UP&DOWN Study DOI Open Access
Ana Gutiérrez-Hervás, Esther Nova, Rocío Izquierdo‐Gómez

et al.

Nutrients, Journal Year: 2024, Volume and Issue: 16(22), P. 3884 - 3884

Published: Nov. 14, 2024

The main objective of this study was to examine the association between lifestyle patterns (physical activity, screen and sleep time diet) abdominal obesity, endocrine, metabolic, immunological biomarkers in adolescents with Down syndrome (DS).

Language: Английский

Citations

0

The Microbiota–Gut–Brain Axis: Key Mechanisms Driving Glymphopathy and Cerebral Small Vessel Disease DOI Creative Commons
Che Mohd Nasril Che Mohd Nassir, Muhammad Danial Che Ramli, Mazira Mohamad Ghazali

et al.

Life, Journal Year: 2024, Volume and Issue: 15(1), P. 3 - 3

Published: Dec. 24, 2024

The human microbiota constitute a very complex ecosystem of microorganisms inhabiting both the inside and outside our bodies, in which health maintenance disease modification are main regulatory features. recent explosion microbiome research has begun to detail its important role neurological health, particularly concerning cerebral small vessel (CSVD), disorder associated with cognitive decline vascular dementia. This narrative review represents state-of-the-art knowledge intimate, interplay between brain through gut-brain axis (GBA) emerging glymphatic system dysfunction (glymphopathy) circulating cell-derived microparticles (MPs) as mediators these interactions. We discuss how microbial dysbiosis promotes neuroinflammation, dysfunction, impaired waste clearance brain, critical factors pathogenesis CSVD. Further, we lifestyle that shape composition functionality microbiota, focusing on sleep modifiable risk factor disorders. presents from neuroscientific perspective establish future therapeutic avenues targeting improve reduce burden

Language: Английский

Citations

0