Integrating psychological and neurobiological considerations regarding the development and maintenance of specific Internet-use disorders: An Interaction of Person-Affect-Cognition-Execution (I-PACE) model

Neuroscience & Biobehavioral Reviews, Journal Year: 2016, Volume and Issue: 71, P. 252 - 266

Published: Sept. 6, 2016

Within the last two decades, many studies have addressed clinical phenomenon of Internet-use disorders, with a particular focus on Internet-gaming disorder. Based previous theoretical considerations and empirical findings, we suggest an Interaction Person-Affect-Cognition-Execution (I-PACE) model specific disorders. The I-PACE is framework for processes underlying development maintenance addictive use certain Internet applications or sites promoting gaming, gambling, pornography viewing, shopping, communication. composed as process model. Specific disorders are considered to be consequence interactions between predisposing factors, such neurobiological psychological constitutions, moderators, coping styles Internet-related cognitive biases, mediators, affective responses situational triggers in combination reduced executive functioning. Conditioning may strengthen these associations within addiction process. Although hypotheses regarding mechanisms summarized model, must further tested empirically, implications treatment interventions suggested.

Language: Английский

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Psychology of Habit

Annual Review of Psychology, Journal Year: 2015, Volume and Issue: 67(1), P. 289 - 314

Published: Sept. 11, 2015

As the proverbial creatures of habit, people tend to repeat same behaviors in recurring contexts. This review characterizes habits terms their cognitive, motivational, and neurobiological properties. In so doing, we identify three ways that interface with deliberate goal pursuit: First, form as pursue goals by repeating responses a given context. Second, outlined computational models, pursuit guide actions synergistically, although are efficient, default mode response. Third, infer from frequency habit performance behavior must have been intended. We conclude applying insights research understand stress addiction well design effective interventions change health consumer behaviors.

Language: Английский

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Drug Addiction: Updating Actions to Habits to Compulsions Ten Years On

Annual Review of Psychology, Journal Year: 2015, Volume and Issue: 67(1), P. 23 - 50

Published: Aug. 8, 2015

A decade ago, we hypothesized that drug addiction can be viewed as a transition from voluntary, recreational use to compulsive drug-seeking habits, neurally underpinned by prefrontal cortical striatal control over seeking and taking well progression the ventral dorsal striatum. Here, in light of burgeoning, supportive evidence, reconsider elaborate this hypothesis, particular refinements our understanding mechanisms underlying goal-directed habitual seeking, influence drug-associated Pavlovian-conditioned stimuli on relapse, evidence for impairments top-down inhibitory behavior. We further review animal human studies have begun define etiological factors individual differences propensity become addicted drugs, leading description endophenotypes, especially cocaine addiction. consider prospect novel treatments promote abstinence relapse use.

Language: Английский

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The transition to compulsion in addiction

Nature reviews. Neuroscience, Journal Year: 2020, Volume and Issue: 21(5), P. 247 - 263

Published: March 30, 2020

Language: Английский

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Synaptic mechanisms underlying persistent cocaine craving

Nature reviews. Neuroscience, Journal Year: 2016, Volume and Issue: 17(6), P. 351 - 365

Published: May 6, 2016

Language: Английский

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Common Brain Mechanisms of Chronic Pain and Addiction

Neuron, Journal Year: 2016, Volume and Issue: 89(1), P. 11 - 36

Published: Jan. 1, 2016

Language: Английский

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Cognitive deficits in individuals with methamphetamine use disorder: A meta-analysis

Addictive Behaviors, Journal Year: 2018, Volume and Issue: 80, P. 154 - 160

Published: Jan. 30, 2018

Language: Английский

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Addiction as a brain disease revised: why it still matters, and the need for consilience

Neuropsychopharmacology, Journal Year: 2021, Volume and Issue: 46(10), P. 1715 - 1723

Published: Feb. 22, 2021

The view that substance addiction is a brain disease, although widely accepted in the neuroscience community, has become subject to acerbic criticism recent years. These criticisms state disease deterministic, fails account for heterogeneity remission and recovery, places too much emphasis on compulsive dimension of addiction, specific neural signature not been identified. We acknowledge some these have merit, but assert foundational premise neurobiological basis fundamentally sound. also emphasize denying harmful standpoint since it contributes reducing access healthcare treatment, consequences which are catastrophic. Here, we therefore address criticisms, doing so provide contemporary update addiction. arguments support this view, discuss why apparently spontaneous does negate it, how seemingly behaviors can co-exist with sensitivity alternative reinforcement Most importantly, argue biological substrate from both capacity behavior change arise, arguing an intensified neuroscientific study recovery. More broadly, propose disagreements reveal need multidisciplinary research integrates neuroscientific, behavioral, clinical, sociocultural perspectives.

Language: Английский

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Epigenetic Mechanisms of Opioid Addiction

Biological Psychiatry, Journal Year: 2019, Volume and Issue: 87(1), P. 22 - 33

Published: July 8, 2019

Language: Английский

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In search of predictive endophenotypes in addiction: insights from preclinical research

Genes Brain & Behavior, Journal Year: 2015, Volume and Issue: 15(1), P. 74 - 88

Published: Oct. 20, 2015

Drug addiction is widely recognized to afflict some but not all individuals by virtue of underlying risk markers and traits involving multifaceted interactions between polygenic external factors. Remarkably, only a small proportion exposed licit illicit drugs develop compulsive drug‐seeking behavior, maintained in the face adverse consequences associated detrimental patterns drug intake extended repeated bouts binge intoxication, withdrawal relapse. As consequence, research has increasingly endeavored identify distinctive neurobehavioral mechanisms endophenotypes that predispose use. However, active users hampered difficulty categorizing putatively causal behavioral prior initiation By contrast, experimental animals often hindered validity approaches used investigate neural psychological habits humans. Herein, we survey discuss principal findings emanating from preclinical animal on highlight how specific presumed genetic origin (e.g. trait anxiety, novelty preference impulsivity) differentially contribute forms seeking taking and, particular, these differentiate different classes stimulant non‐stimulant abuse.

Language: Английский

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