Addictive Behaviors Reports,
Journal Year:
2015,
Volume and Issue:
2, P. 41 - 48
Published: June 6, 2015
It
has
been
suggested
that
addictive
behaviors
related
to
consumption
of
specific
foods
could
contribute
overeating
and
obesity.
Although
energy-dense,
hyper-palatable
are
hypothesized
be
associated
with
addictive-like
eating
behaviors,
few
studies
have
assessed
this
in
humans.
To
evaluate
young
adults
whether
intakes
'food
addiction',
as
by
the
Yale
Food
Addiction
Scale
(YFAS),
describe
nutrient
intake
profiles.
Australian
aged
18–35
years
were
invited
complete
an
online
cross-sectional
survey
including
demographics,
YFAS
usual
dietary
intake.
Participants
classified
food
addicted
(FAD)
or
non-addicted
(NFA)
according
predefined
scoring
criteria.
A
total
462
participants
(86%
female,
73%
normal
weight)
completed
survey,
14.7%
(n
=
68)
FAD.
The
FAD
group
had
a
higher
proportion
females
(p
.01)
body
mass
index
<
.001)
compared
NFA.
Higher
symptom
scores
percentage
energy
(%E)
from
nutrient-poor
candy,
take
out
baked
sweet
products,
well
lower
%E
nutrient-dense
core
whole-grain
products
breakfast
cereals.
These
remained
statistically
significant
when
adjusted
for
age,
sex
BMI
category
.001).
Statistically
associations
identified
between
addiction
intake,
specifically
foods.
However,
effect
sizes
small
limiting
clinical
applications.
Further
examination
relationship
nationally
representative
sample
is
warranted.
Obesity Facts,
Journal Year:
2015,
Volume and Issue:
8(6), P. 386 - 401
Published: Jan. 1, 2015
The
proposition
of
so-called
‘food
addiction'
(FA)
in
the
scientific
literature
has
stimulated
a
recent
surge
research
and
debate.
concept
FA
is
controversial,
opinion
divided.
Many
findings
depend
upon
use
single
instrument
called
Yale
Food
Addiction
Scale
(YFAS).
This
review
systematically
examined
FA,
as
defined
by
YFAS,
reported
40
experimental
human
studies
published
or
after
2009.
results
indicated
that
much
makes
supposition
food
addiction
an
accepted
neurobiological
disease,
consistent
with
substance
disorders;
interpretation
based
on
very
limited
data.
raises
question
to
whether
those
individuals
who
meet
YFAS
criteria
for
diagnosis
are
truly
‘addicted'
if
they
experience
significant
impairment
their
psychological
wellbeing
quality
life
would
be
expected
clinically
recognised
addictive
disorders.
At
present
time,
little
investigated
extent
which
psychometric
self-assessment
symptomatology
can
elucidate
harmful
relationship
target
foods
diet.
A
positive
usually
positively
associated
BMI
strongly
linked
presence
binge
eating,
but
certain
exceptions
within
were
revealed.
Further
clarification
required
sufficiently
different
existing
conditions
traits
warrant
classification
distinctive
disease
phenotype
rather
than
expression
strong
habits
preferences.
Neuroscience & Biobehavioral Reviews,
Journal Year:
2016,
Volume and Issue:
63, P. 223 - 238
Published: Feb. 2, 2016
Relatively
little
is
known
about
the
neuropathophysiology
of
binge-eating
disorder
(BED).
Here,
evidence
from
neuroimaging,
neurocognitive,
genetics,
and
animal
studies
are
reviewed
to
synthesize
our
current
understanding
pathophysiology
BED.
Binge-eating
may
be
conceptualized
as
an
impulsive/compulsive
disorder,
with
altered
reward
sensitivity
food-related
attentional
biases.
Neuroimaging
suggest
there
corticostriatal
circuitry
alterations
in
BED
similar
those
observed
substance
abuse,
including
function
prefrontal,
insular,
orbitofrontal
cortices
striatum.
Human
genetics
that
changes
neurotransmitter
networks,
dopaminergic
opioidergic
systems,
associated
behaviors.
Overall,
suggests
related
maladaptation
regulating
motivation
impulse
control
found
other
disorders.
Further
needed
understand
how
activity
neurocircuitry
pharmacotherapies
influence
these
systems
reduce
symptoms.
Nutrients,
Journal Year:
2018,
Volume and Issue:
10(4), P. 477 - 477
Published: April 12, 2018
The
diagnostic
construct
of
“food
addiction”
is
a
highly
controversial
subject.
current
systematic
review
the
first
to
evaluate
empirical
studies
examining
in
humans
and
animals.
Studies
were
included
if
they
quantitative,
peer-reviewed,
English
language.
52
identified
(35
articles)
qualitatively
assessed
determine
extent
which
their
findings
indicated
following
addiction
characteristics
relation
food:
brain
reward
dysfunction,
preoccupation,
risky
use,
impaired
control,
tolerance/withdrawal,
social
impairment,
chronicity,
relapse.
Each
pre-defined
criterion
was
supported
by
at
least
one
study.
Brain
dysfunction
control
largest
number
(n
=
21
n
12,
respectively);
whereas
use
fewest
1).
Overall,
support
food
as
unique
consistent
with
criteria
for
other
substance
disorder
diagnoses.
evidence
further
suggests
that
certain
foods,
particularly
processed
foods
added
sweeteners
fats,
demonstrate
greatest
addictive
potential.
Though
both
behavioral
substance-related
factors
are
implicated
process,
symptoms
appear
better
fit
than
addiction.
Future
research
should
explore
social/role
associated
potential
interventions
prevention
treatment.
Nutrients,
Journal Year:
2017,
Volume and Issue:
9(11), P. 1170 - 1170
Published: Oct. 27, 2017
The
specific
eating
pattern
of
Binge
Eating
Disorder
(BED)
patients
has
provoked
the
assumption
that
BED
might
represent
a
phenotype
within
obesity
spectrum
is
characterized
by
increased
impulsivity.
Following
guidelines
PRISMA
statement
(preferred
reporting
items
for
systematic
reviews
and
meta-analyses),
we
here
provide
update
on
evidence
food-related
impulsivity
in
obese
individuals,
with
without
BED,
as
well
normal-weight
individuals.
We
separately
analyzed
potential
group
differences
components
reward
sensitivity
rash-spontaneous
behavior.
Our
search
resulted
twenty
experimental
studies
high
methodological
quality.
synthesis
latest
consolidates
conclusions
drawn
our
initial
review
represents
distinct
Rash-spontaneous
behavior
general,
specifically
towards
food,
while
food-specific
also
individuals
but
potentially
to
lesser
degree.
A
major
next
step
research
entails
investigation
sub-domains
temporal
inhibitory
control
obesity.
Based
impaired
affected
profit
from
interventions
address
impulsive
Journal of Human Nutrition and Dietetics,
Journal Year:
2018,
Volume and Issue:
31(4), P. 544 - 572
Published: Jan. 25, 2018
Abstract
Background
The
present
study
systematically
reviewed
the
literature
aiming
to
determine
relationships
between
food
addiction,
as
measured
by
Yale
Food
Addiction
Scale
(
YFAS
),
and
mental
health
symptoms.
Methods
Nine
databases
were
searched
using
keywords.
Studies
included
if
they
reported:
(i)
diagnosis
or
symptom
score
(ii)
a
outcome,
well
association
(ii).
In
total,
51
studies
included.
Results
Through
meta‐analysis,
mean
prevalence
of
addiction
was
16.2%,
with
an
average
3.3
(range
2.85–3.92)
symptoms
being
reported.
Subanalyses
revealed
that
number
in
populations
seeking
treatment
for
weight
loss
3.01
2.65–3.37)
this
higher
groups
disordered
eating
(mean
5.2
3.6–6.7).
Significant
positive
correlations
found
binge
[mean
r
=
0.602
(0.557–0.643),
P
<
0.05],
depression,
anxiety
0.459
(0.358–0.550),
0.483
(0.228–0.676),
0.05,
respectively].
Conclusions
A
significant,
relationship
exists
symptoms,
although
results
highlight
complexity
relationship.
Neuroscience & Biobehavioral Reviews,
Journal Year:
2019,
Volume and Issue:
103, P. 178 - 199
Published: May 21, 2019
In
2016
the
World
Health
Organization
reported
39%
of
world's
adult
population
(over
18
y)
was
overweight,
with
western
countries
such
as
Australia
and
United
States
America
at
64.5%
67.9%
respectively.
Overconsumption
high
fat/sugar
containing
food
beverages
contribute
to
development
obesity.
Neural
plasticity
that
occurs
a
result
long
term
sugar
consumption
has
been
shown
reduce
impulse
control
therefore
lower
ability
resist
foods
contributing
obesity
epidemic.
There
is
significant
overlap
between
neural
pathways
involved
in
emotions
guide
behavioural
responses
survival
situations
those
regulating
overconsumption
highly
palatable
food.
This
suggests
having
clearer
understanding
role
stress
will
lead
novel
therapeutic
strategies.
Sucrose
activates
mesocorticolimbic
system
manner
synonymous
substances
abuse.
overwhelming
evidence
support
hypothesis
sucrose
results
pathophysiological
consequences
morphological
neuronal
changes,
altered
emotional
processing
modified
behaviour
rodent
human
models.
this
comprehensive
review,
we
examined
>300
studies
investigating
interaction
consumption,
emotions.
Preclinical
clinical
trials
stress,
anxiety,
depression
fear
are
reviewed.
Importantly,
synergy
neurobiology
addressed.
review
summarizes
neurochemical
changes
adaptations
ö
including
dopaminergic
influence
emotion
following
consumption.